Fumio Hanaoka - Genetic Mutations

Author Information

Fumio Hanaoka is a distinguished researcher affiliated with multiple prestigious institutions in Japan. His primary affiliations include the Department of Life Science at the Faculty of Science, Gakushuin University in Tokyo; the Graduate School of Frontier Biosciences at Osaka University in Osaka; and the National Institute of Genetics in Shizuoka. His research spans various aspects of genetics, with a particular focus on DNA polymerases and DNA repair mechanisms.

Research Contributions

Fumio Hanaoka has made significant contributions to the field of genetics, particularly in understanding the mechanisms of DNA repair and replication. His recent papers delve into topics such as the interaction of DNA polymerase alpha with telomeres, the effects of oxatomide derivatives on human receptors, and the role of the ubiquitin-proteasome system in DNA damage recognition. His work has provided valuable insights into the cellular response to genotoxic stress and the maintenance of genomic stability.

Aliases

Fumio Hanaoka is also known by several aliases in academic publications, including F. Hanaoka, F Hanaoka, F E Hanaoka, and Fumi Hanaoka.

Publication and Citation Metrics

Metric	Value
Citation Count	24,381
h-index	78
Paper Count	520

Publications:

The intrinsically disordered N‐terminal region of mouse DNA polymerase alpha mediates its interaction with POT1a/b at telomeres

DOI: 10.1111/gtc.12845

Year: 2021

Inhibitory effects of oxatomide derivatives on human P2X7 receotor

DOI:

Year: 2020

Functional impacts of the ubiquitin–proteasome system on DNA damage recognition in global genome nucleotide excision repair

DOI: 10.1038/s41598-020-76898-2

Year: 2020

Effect of sequence context on Polζ-dependent error-prone extension past (6-4) photoproducts.

DOI: 10.1016/j.dnarep.2019.102771

Year: 2019

Replication stress triggers microsatellite destabilization and hypermutation leading to clonal expansion in vitro

DOI: 10.1038/s41467-019-11760-2

Year: 2019

Analysis of DNA replication inhibition and mutagenesis induced by N 7-glycidamide-dG

DOI:

Year: 2019

Analysis of interaction between DNA polymerase η and a deubiquitinating enzyme

DOI:

Year: 2019

Kobe University Repository : Kernel タイトル Tit le SUMOylat ion of xeroderma pigmentosum group C protein regulates DNA damage recognit ion during nucleot ide excision repair

DOI:

Year: 2018

Hypersensitivity of mouse embryonic fibroblast cells defective for DNA polymerases η, ι and κ to various genotoxic compounds: Its potential for application in chemical genotoxic screening.

DOI: 10.1016/j.dnarep.2017.11.006

Year: 2018

Polη, a Y-family translesion synthesis polymerase, promotes cellular tolerance of Myc-induced replication stress

DOI: 10.1242/jcs.212183

Year: 2018

How Do Microarrays Work?

What Are the Challenges in Phylogenetic Analysis?

How Does the Body Repair UV-Induced DNA Damage?

What is Cancer Genomics?

What Types of Cancers are Associated with BRCA1 and BRCA2 Mutations?

What Are the Future Directions in Sickle Cell Disease Research?

What Are Some Examples of Genetic Disorders Caused by Mutations?

Are there preventive measures for PAH exposure?

What are TALENs?

What Makes a Mutation Harmful?

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