Guillermo Zalba - Genetic Mutations

Author Information

Guillermo Zalba is a distinguished researcher affiliated with the Instituto de Investigación Sanitaria de Navarra and the Department of Biochemistry and Genetics at the University of Navarra in Pamplona, Spain. His work primarily focuses on the intricate aspects of genetics and biochemistry, contributing significantly to the understanding of cardiovascular health, oxidative stress, and vascular pathophysiology.

Research Contributions

Guillermo Zalba's research contributions are notable in the fields of cardiovascular health and genetics, with a particular interest in the mechanisms of oxidative stress and its impact on vascular diseases. His studies have explored various dimensions such as the relationship between dietary exposure to pollutants and telomere length, the role of hemostatic parameters in stroke outcomes, and the influence of endothelial NADPH oxidase activity on metabolic processes. His work extends to studying the effects of oxidative stress on the blood-brain barrier and memory, as well as the implications of endothelial dysfunction in atherosclerosis and aneurysms.

Aliases

Guillermo Zalba is also known by several aliases in scientific literature, including G Zalba and G. Zalba.

Publication and Citation Metrics

Metric	Value
Total Citation Count	4617
h-index	39
Total Paper Count	143

Publications:

Dietary Exposure to Polychlorinated Biphenyls and Dioxins and Its Relationship to Telomere Length in Subjects Older Than 55 Years from the SUN Project

DOI: 10.3390/nu14020353

Year: 2022

Association between ideal cardiovascular health and telomere length in participants older than 55 years old from the SUN cohort.

DOI: 10.1016/j.rec.2021.04.002

Year: 2021

Inside the Thrombus: Association of Hemostatic Parameters With Outcomes in Large Vessel Stroke Patients

DOI: 10.3389/fneur.2021.599498

Year: 2021

Oxidative Stress in Vascular Pathophysiology: Still Much to Learn

DOI: 10.3390/antiox10050673

Year: 2021

Expression of Endothelial NOX5 Alters the Integrity of the Blood-Brain Barrier and Causes Loss of Memory in Aging Mice

DOI: 10.3390/antiox10081311

Year: 2021

Endothelial NOX5 Expression Modulates Thermogenesis and Lipolysis in Mice Fed with a High-Fat Diet and 3T3-L1 Adipocytes through an Interleukin-6 Dependent Mechanism

DOI: 10.3390/antiox11010030

Year: 2021

NADPH Oxidase 5 Induces Changes in the Unfolded Protein Response in Human Aortic Endothelial Cells and in Endothelial-Specific Knock-in Mice

DOI: 10.3390/antiox10020194

Year: 2021

Interferon stimulated gene 15 pathway is a novel mediator of endothelial dysfunction and aneurysms development in angiotensin II infused mice through increased oxidative stress.

DOI: 10.1093/cvr/cvab321

Year: 2021

Endothelial Nox5 Expression Modulates Glucose Uptake and Lipid Accumulation in Mice Fed a High-Fat Diet and 3T3-L1 Adipocytes Treated with Glucose and Palmitic Acid

DOI: 10.3390/ijms22052729

Year: 2021

NADPH Oxidase Overactivity Underlies Telomere Shortening in Human Atherosclerosis

DOI: 10.3390/ijms21041434

Year: 2020

What Can Karyotyping Detect?

Can Genetic Mutations be Treated or Cured?

How do Duplications Occur?

Can Genetic Mutations Be Treated or Prevented?

What Are the Different Types of Mutations?

What Tools are Used to Analyze CNVs?

What are the Technical Challenges?

What is the Role of Neutral Mutations in Evolution?

How Do Beneficial Mutations Spread in Populations?

What Ethical Considerations Are Involved?

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