Kiyoshi Takatsu - Genetic Mutations

Author Information

Kiyoshi Takatsu is a distinguished researcher affiliated with the Department of Immunobiology and Pharmacological Genetics at the Graduate School of Medicine and Pharmaceutical Science for Research, University of Toyama, in Toyama, Japan. His work primarily focuses on the fields of immunobiology and pharmacological genetics, making significant contributions to the understanding of immune responses and their implications for human health.

Research Contributions

Kiyoshi Takatsu has made substantial contributions to the field of genetics and immunology, with a particular emphasis on understanding the mechanisms of allergic inflammation and metabolic disorders. His research has explored the roles of cytokines such as interleukin-5 (IL-5) in allergic diseases and the immune system's interactions with metabolic processes, as evidenced by his studies on the modulation of gut bacteria and inflammation in adipose tissue. His recent work includes exploring the effects of natural compounds like betulin and isoliquiritigenin on immune activity and metabolic health.

Aliases

Kiyoshi Takatsu is also known by the aliases K Takatsu and K. Takatsu. These variations of his name may appear in different publications and databases.

Publication and Citation Metrics

Metric	Value
Total Citations	21,487
h-index	75
Total Number of Papers	446

Publications:

Betulin Attenuates TGF-β1- and PGE2-Mediated Inhibition of NK Cell Activity to Suppress Tumor Progression and Metastasis in Mice.

DOI: 10.1248/bpb.b21-00921

Year: 2022

Isoliquiritigenin attenuates adipose tissue inflammation and metabolic syndrome by modifying gut bacteria composition in mice.

DOI: 10.1002/mnfr.202101119

Year: 2022

Toyama's Pharmaceuticals : Its Origin and Drug Development

DOI: 10.4325/seikeikakou.33.200

Year: 2021

Bidirectional crosstalk between neutrophils and adipocytes promotes adipose tissue inflammation

DOI: 10.1096/fj.201900477RR

Year: 2019

Metabolism and biochemical properties of nicotinamide adenine dinucleotide (NAD) analogs, nicotinamide guanine dinucleotide (NGD) and nicotinamide hypoxanthine dinucleotide (NHD)

DOI: 10.1038/s41598-019-49547-6

Year: 2019

[IL-5 AND ALLERGIC INFLAMMATION].

DOI: 10.15036/arerugi.67.108

Year: 2018

Elimination of eosinophils using anti-IL-5 receptor alpha antibodies effectively suppresses IL-33-mediated pulmonary arterial hypertrophy.

DOI: 10.1016/j.imbio.2017.12.002

Year: 2018

CD206+ M2-like macrophages regulate systemic glucose metabolism by inhibiting proliferation of adipocyte progenitors

DOI: 10.1038/s41467-017-00231-1

Year: 2017

Allergic diseases: From bench to clinic - Contribution of the discovery of interleukin-5.

DOI: 10.1016/j.cyto.2016.11.011

Year: 2017

Prolonged activation of IL-5-producing ILC2 causes pulmonary arterial hypertrophy.

DOI: 10.1172/jci.insight.90721

Year: 2017

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What Types of Radiation Are Most Harmful?

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What Types of Cancers are Associated with BRCA1 and BRCA2 Mutations?

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How Does PacBio's SMRT Technology Work?

What is Prenatal Screening?

What are the Signs of BRCA1 and BRCA2 Mutations?

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