Mohammad Abdulmohsen Alfhili - Genetic Mutations

Author Information

Mohammad Abdulmohsen Alfhili is affiliated with the Chair of Medical and Molecular Genetics Research, Department of Clinical Laboratory Sciences, College of Applied Medical Sciences, King Saud University, Riyadh, Saudi Arabia. His work primarily focuses on various aspects of molecular genetics and clinical laboratory sciences.

Research Contributions

Mohammad Abdulmohsen Alfhili has made significant contributions to the field of molecular genetics and clinical laboratory sciences. His research spans a variety of topics, including the mechanisms of premature red blood cell death (eryptosis), molecular pathways involved in hemolysis, and the impact of various compounds on blood cells. Noteworthy among his publications are studies on the effects of geraniin on whole blood cytokines and erythrocyte death, the induction of hemolysis by occupational pollutants like nickel chloride, and the apoptotic effects of triclosan on lymphoma cells. His work often highlights the role of calcium signaling and oxidative stress in cellular processes.

Aliases

Mohammad Abdulmohsen Alfhili is also known by the following aliases: Mohammad A Alfhili, Mohammad A. Alfhili, and Mohammad Alfhili.

Publication and Citation Metrics

Metric	Value
Citation Count	432
h-index	11
Paper Count	59

Publications:

Geraniin inhibits whole blood IFN-γ and IL-6 and promotes IL-1β and IL-8, and stimulates calcium-dependent and sucrose-sensitive erythrocyte death

DOI: 10.1016/j.taap.2022.115881

Year: 2022

Flow Cytofluorometric Analysis of Molecular Mechanisms of Premature Red Blood Cell Death.

DOI: 10.1007/978-1-0716-1514-0_11

Year: 2021

Induction of hemolysis and eryptosis by occupational pollutant nickel chloride is mediated through calcium influx and p38 MAP kinase signaling.

DOI: 10.13075/ijomeh.1896.01814

Year: 2021

Triclosan induces apoptosis in Burkitt lymphoma-derived BJAB cells through caspase and JNK/MAPK pathways

DOI: 10.1007/s10495-020-01650-0

Year: 2021

The soma-germline communication: implications for somatic and reproductive aging

DOI: 10.5483/BMBRep.2021.54.5.198

Year: 2021

Epidemic dropsy toxin, sanguinarine chloride, stimulates sucrose-sensitive hemolysis and breakdown of membrane phospholipid asymmetry in human erythrocytes.

DOI: 10.1016/j.toxicon.2021.05.013

Year: 2021

Synergistic efficacies of thymoquinone and standard antibiotics against multi-drug resistant isolates

DOI: 10.15537/smj.2021.2.25706

Year: 2021

Reprogramming of erythrocyte lifespan by NFκB-TNFα naphthoquinone antagonist β-lapachone is regulated by calcium overload and CK1α.

DOI: 10.1111/jfbc.13710

Year: 2021

Calcium-oxidative stress signaling axis and casein kinase 1α mediate eryptosis and hemolysis elicited by novel p53 agonist inauhzin.

DOI: 10.1080/1120009X.2021.1963616

Year: 2021

Physcion induces hemolysis and premature phosphatidylserine externalization in human erythrocytes.

DOI: 10.1248/bpb.b20-00744

Year: 2021

What is Cellular Heterogeneity?

What is Cystic Fibrosis (CF)?

How Do Genetic Mutations Contribute to Heart Disease?

Can Gene Amplification Be Targeted Therapeutically?

What is Genetic Research?

How is the Delta F508 Mutation Diagnosed?

What Types of Mutations Are Commonly Observed?

What Are the Successes of Gene Therapy?

How Are Genetic Diseases Diagnosed?

What Role Does Genetic Testing Play in Managing Infectious Diseases?

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