Yun-dong Bai - Genetic Mutations

Author Information

Yun Bai is a prominent researcher affiliated with the Department of Medical Genetics, College of Basic Medical Science, Third Military Medical University, located in Chongqing, China. His work primarily focuses on the intricate mechanisms of bone formation, osteoclastogenesis, and genetic factors influencing bone diseases and related conditions.

Research Contributions

Yun Bai has made significant contributions to the field of genetics and bone biology. His research explores various aspects of bone healing and regeneration, with a particular focus on the roles of long non-coding RNAs (lncRNAs), inflammatory processes, and genetic screening strategies. Notable works include studies on the effects of lncRNA on bone repair, the influence of osteoclast-derived exosomes on osteoblasts, as well as the investigation into the genetic underpinnings of intellectual disabilities. His research also delves into the molecular pathways involved in osteoclastogenesis and the potential therapeutic implications of bioceramics and graphene-based technologies in enhancing bone regeneration.

Aliases

Yun Bai is also known by the alias Yun-dong Bai in various publications and research activities.

Publication and Citation Metrics

Metric	Count
Publication Count	138
Citation Count	4143
H-index	34

Publications:

Long non-coding RNA HCAR promotes endochondral bone repair by upregulating VEGF and MMP13 in hypertrophic chondrocyte through sponging miR-15b-5p

DOI: 10.1016/j.gendis.2020.07.013

Year: 2022

Inflammatory osteoclasts‐derived exosomes promote bone formation by selectively transferring lncRNA LIOCE into osteoblasts to interact with and stabilize Osterix

DOI: 10.1096/fj.202101106RR

Year: 2022

X Chromosome Inactivation Pattern and Pregnancy Outcome of Female Carriers of Pathogenic Heterozygous X-Linked Deletions

DOI: 10.3389/fgene.2021.782629

Year: 2021

Connective Tissue Growth Factor From Periosteal Tartrate Acid Phosphatase-Positive Monocytes Direct Skeletal Stem Cell Renewal and Fate During Bone Healing

DOI: 10.3389/fcell.2021.730095

Year: 2021

Klotho upregulates the interaction between RANK and TRAF6 to facilitate RANKL-induced osteoclastogenesis via the NF-κB signaling pathway

DOI: 10.21037/atm-21-4332

Year: 2021

A Tiered Genetic Screening Strategy for the Molecular Diagnosis of Intellectual Disability in Chinese Patients

DOI: 10.3389/fgene.2021.669217

Year: 2021

Graphene‐Based MicroRNA Transfection Blocks Preosteoclast Fusion to Increase Bone Formation and Vascularization

DOI: 10.1002/advs.202102286

Year: 2021

Mn-containing bioceramics inhibit osteoclastogenesis and promote osteoporotic bone regeneration via scavenging ROS

DOI: 10.1016/j.bioactmat.2021.03.039

Year: 2021

Chondrogenesis mediates progression of ankylosing spondylitis through heterotopic ossification

DOI: 10.1038/s41413-021-00140-6

Year: 2021

Inhibition of Lactate-GPR81-PI3K/Akt Pathway May Exacerbate Aβ Aggregation in 3-Month-Old APP/PS1 Mice

DOI: 10.21203/rs.3.rs-651317/v1

Year: 2021

Can Genetic Testing Help?

What is the Prognosis for Individuals with Cystic Fibrosis?

What Are Some Examples of Pharmacogenomic Applications?

What are the Global Implications?

How Do Genetic Mutations Occur?

What is Genetic Engineering?

How are Missense Mutations Detected?

What is the future of research in inherited genetic mutations?

How is Cancer Genomics Studied?

What are Acridine Dyes?

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