Hamid Ghaedi - Genetic Mutations

Author Information

Hamid Ghaedi is affiliated with the Department of Medical Genetics at the School of Medicine, Shahid Beheshti University of Medical Sciences, Tehran, Iran. His research spans several areas of genetics, with a focus on molecular subtyping of cancers, genetic variants associated with diseases, and the role of long non-coding RNAs in disease pathogenesis.

Research Contributions

Hamid Ghaedi has made significant contributions to the field of medical genetics. His research includes the study of mutation profiles in muscle invasive bladder cancer, molecular subtyping of bladder cancer using immunohistochemical assays, and the association of genetic variants with coronary artery disease and other conditions. He has also investigated the expression of non-coding RNAs in various diseases, including acute leukemia, coronary artery disease, and type 2 diabetes mellitus. His work on genetic variants in autism and schizophrenia, as well as the analysis of miRNA and gene expression in glioblastoma, highlights his broad expertise in genetic research.

Aliases

Hamid Ghaedi is also known by the aliases H Ghaedi and H Z Ghaedi.

Publication and Citation Metrics

Total Citation Count	970
H-Index	20
Total Number of Papers	86

Publications:

Mutually exclusive mutation profiles define functionally related genes in muscle invasive bladder cancer

DOI: 10.1371/journal.pone.0259992

Year: 2022

Immunohistochemical Assays for Bladder Cancer Molecular Subtyping: Optimizing Parsimony and Performance of Lund Taxonomy Classifiers.

DOI: 10.1369/00221554221095530

Year: 2022

Association of rs2954029 and rs6982502 Variants with Coronary Artery Disease by HRM Technique: A GWAS Replication Study in an Iranian Population.

DOI: 10.52547/rbmb.10.4.580

Year: 2022

Up-regulation of FOXN3-AS1 in invasive ductal carcinoma of breast cancer patients

DOI: 10.1016/j.heliyon.2021.e08179

Year: 2021

Immunohistochemical assays for bladder cancer molecular subtyping: Optimizing parsimony and performance using Lund taxonomy

DOI: 10.1101/2021.08.06.455345

Year: 2021

IFNG-AS1 and MAF4 long non-coding RNAs are upregulated in acute leukemia patients who underwent bone marrow transplantation.

DOI: 10.1016/j.retram.2021.103307

Year: 2021

Variation in the expression level of MALAT1, MIAT and XIST lncRNAs in coronary artery disease patients with and without type 2 diabetes mellitus.

DOI: 10.1080/13813455.2020.1768410

Year: 2020

Hsa‐miR‐27a‐3p and epidermal growth factor receptor expression analysis in glioblastoma FFPE samples

DOI: 10.1111/ajco.13399

Year: 2020

SUCLG1 mutations and mitochondrial encephalomyopathy: a case study and review of the literature.

DOI: 10.1007/s11033-020-05999-y

Year: 2020

Association of rs3735025 and rs9656169 variants with autism, and schizophrenia: A GWAS-replication study in an Iranian population

DOI: 10.1016/j.mgene.2020.100784

Year: 2020

What is Genetic Counseling?

What is Genetic Coding?

How does Pharmacogenomics Relate to Genetic Mutations?

How do Viral Vectors Work?

What are the Technical Challenges?

Are Synonymous Mutations Considered in Genetic Research?

What Role Does Biotechnology Play in Agriculture?

Can Genetic Susceptibility Influence Mutation Risk?

What is the Future of Third Generation Sequencing?

What Causes Cystic Fibrosis?

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