Ituro Inoue - Genetic Mutations

Author Information

Ituro Inoue is affiliated with the Human Genetics Laboratory, Department of Genomics and Evolutionary Biology at the National Institute of Genetics in Mishima, Shizuoka, Japan. His work primarily focuses on various aspects of genetics and genomics, contributing significantly to the understanding of genetic diversity, disease mechanisms, and evolutionary biology.

Research Contributions

Ituro Inoue has made substantial contributions to the field of genetics, with a focus on both human health and disease. His recent publications cover a wide array of topics including the genetic underpinnings of gout, the dynamics of clonal selection in endometrial epithelium, and the role of ancient human endogenous retroviruses. Notably, he has also delved into the genetic diversity of killer immunoglobulin-like receptors and the mutagenic processes driving endometriosis. His work is recognized for its breadth and depth, spanning from detailed genetic analyses to the exploration of complex genetic interactions in various health conditions.

Aliases

Ituro Inoue is also known by several aliases including I Inoue and I. Inoue, but his full name Ituro Inoue is most appropriate for formal references.

Publication and Citation Metrics

Metric	Value
Total Citation Count	7576
h-index	48
Total Paper Count	206

Publications:

Genome-wide meta-analysis between renal overload type and renal underexcretion type of clinically defined gout in Japanese populations.

DOI: 10.1016/j.ymgme.2022.01.100

Year: 2022

Spatiotemporal dynamics of clonal selection and diversification in normal endometrial epithelium

DOI: 10.1038/s41467-022-28568-2

Year: 2022

Movements of Ancient Human Endogenous Retroviruses Detected in SOX2-Expressing Cells

DOI: 10.1128/jvi.00356-22

Year: 2022

Decoding the diversity of killer immunoglobulin-like receptors by deep sequencing and a high-resolution imputation method

DOI: 10.1016/j.xgen.2022.100101

Year: 2022

APOBEC mediated mutagenesis drives genomic heterogeneity in endometriosis.

DOI: 10.1038/s10038-021-01003-y

Year: 2022

Comprehensive discovery of CRISPR-targeted terminally redundant sequences in the human gut metagenome: Viruses, plasmids, and more

DOI: 10.1371/journal.pcbi.1009428

Year: 2021

Significance of Mitochondrial DNA Haplogroup on Epidermal Growth Factor Receptor Mutation in Japanese Patients With Lung Adenocarcinoma

DOI: 10.21873/anticanres.15197

Year: 2021

Substantial anti-gout effect conferred by common and rare dysfunctional variants of URAT1/SLC22A12

DOI: 10.1093/rheumatology/keab327

Year: 2021

Genetic variant rs10251977 (G>A) in EGFR-AS1 modulates the expression of EGFR isoforms A and D

DOI: 10.1038/s41598-021-88161-3

Year: 2021

Proposing a molecular classification associated with hypercoagulation in ovarian clear cell carcinoma.

DOI: 10.1016/j.ygyno.2021.08.009

Year: 2021

What Are the Consequences of Gene Amplification?

How is Benzene Exposure Related to Genetic Mutations?

What are the Ethical Concerns?

What is Phylogenetic Analysis?

What are the Common Hemoglobin Variants Detected?

How Does Gene Amplification Occur?

What are Neurodevelopmental Disorders?

Why is Long-Read Sequencing Important for Genetic Mutations?

How are Patients Monitored During TKI Therapy?

What Causes Sickle Cell Disease?

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