Kimi Araki - Genetic Mutations

Author Information

Kimi Araki is a prominent researcher affiliated with the Laboratory of Developmental Genetics at the Institute of Resource Development and Analysis, located in Chuo-Ku, Honjo 2-2-1, Kumamoto, Japan. This affiliation reflects a strong background in developmental genetics, contributing to various fields within genetics research.

Research Contributions

Kimi Araki has made significant contributions to the field of genetics, as demonstrated by a prolific publication record. The research topics span a range of genetic studies, including the control of neocortical layers by thalamocortical axons, the role of the acidic domain of Hmga2 in hematopoietic stem cell self-renewal, and the importance of FBXO47 in mouse male meiosis. Additional key studies include the investigation of skin tumor susceptibility related to Pak1-3'UTR polymorphism, resistance to chemical carcinogenesis in naked mole-rats, and the role of lncRNA Caren in heart failure. Araki's diverse research portfolio highlights a deep engagement with both fundamental and applied genetics.

Aliases

Kimi Araki is also known by the aliases "K. Araki" and "K Araki."

Publication and Citation Metrics

Metric	Value
Citation Count	8635
H-index	47
Paper Count	257

Publications:

Thalamocortical axons control the cytoarchitecture of neocortical layers by area-specific supply of VGF

DOI: 10.7554/eLife.67549

Year: 2022

The acidic domain of Hmga2 and the domain’s linker region are critical for driving self-renewal of hematopoietic stem cell

DOI: 10.1007/s12185-021-03274-9

Year: 2022

FBXO47 is essential for preventing the synaptonemal complex from premature disassembly in mouse male meiosis

DOI: 10.1016/j.isci.2022.104008

Year: 2022

Functional polymorphism in Pak1-3'UTR alters skin tumor susceptibility by alternative polyadenylation.

DOI: 10.1016/j.jid.2022.02.009

Year: 2022

Resistance to chemical carcinogenesis induction via a dampened inflammatory response in naked mole-rats

DOI: 10.1038/s42003-022-03241-y

Year: 2022

The lncRNA Caren antagonizes heart failure by inactivating DNA damage response and activating mitochondrial biogenesis

DOI: 10.1038/s41467-021-22735-7

Year: 2021

FBXO47 is essential for preventing the synaptonemal complex from premature disassembly in mouse male meiosis

DOI: 10.1101/2021.11.17.469052

Year: 2021

HMGA2 Maintains Hematopoietic Stem Cell Via Pleiotropic Regulation of the Transcription in Stress Conditions

DOI: 10.1182/blood-2021-152472

Year: 2021

FBXO47 Is Essential for Preventing the Synaptonemal Complex From Premature Disassembly in Mouse Male Meiosis

DOI: 10.2139/ssrn.3971109

Year: 2021

The polyol pathway is an evolutionarily conserved system for sensing glucose uptake

DOI: 10.1101/2021.09.14.460366

Year: 2021

What Are the Ethical Implications of Genetic Testing?

What Is the Mechanism Behind Asbestos-Induced Mutagenesis?

What Are the Different Types of Mutations?

What is Gene Cloning?

What Types of Genetic Mutations Can Benzene Cause?

How do Genetic Mutations Lead to Cancer?

Are All Silent Mutations Neutral?

What is the Role of Genetics in Cancer Treatment?

What is the Future of Genetic Mutations in Agriculture?

What is the Future of CFTR Gene Research?

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