Hitoshi Niwa - Genetic Mutations

Author Information

Hitoshi Niwa is affiliated with the Department of Pluripotent Stem Cell Biology at the Institute of Molecular Embryology and Genetics (IMEG), Kumamoto University, located at 2-2-1 Honjo, Chuo-ku, Kumamoto, 860-0811, Japan. Additionally, he is associated with the Liaison Laboratory Research Promotion Center at the same institution. His research primarily focuses on pluripotent stem cell biology and molecular embryology.

Research Contributions

Hitoshi Niwa has made significant contributions to the field of genetics and stem cell research. In 2021, he published several papers on topics including the polyol pathway as a glucose sensor, meiosis-specific repressor complexes in spermatogenesis, and the promotion of naive pluripotency in stem cells. His research has also delved into the establishment of self-renewing macrophages, the transition from mitosis to meiosis in germ cells, and the molecular detection of kidney maturation stages. His work is characterized by a focus on the molecular mechanisms governing cell differentiation and development.

Aliases

Hitoshi Niwa is also known by the aliases H. Niwa and H Niwa.

Publication and Citation Metrics

Metric	Value
Citation Count	25678
h-index	60
Paper Count	163

Publications:

The polyol pathway is an evolutionarily conserved system for sensing glucose uptake

DOI: 10.1101/2021.09.14.460366

Year: 2021

Meiosis-specific ZFP541 repressor complex promotes meiotic prophase exit during spermatogenesis

DOI: 10.1101/2021.01.15.426901

Year: 2021

Meiosis-specific ZFP541 repressor complex promotes developmental progression of meiotic prophase towards completion during mouse spermatogenesis

DOI: 10.1038/s41467-021-23378-4

Year: 2021

Inhibition of N-myristoyltransferase Promotes Naive Pluripotency in Mouse and Human Pluripotent Stem Cells

DOI: 10.1101/2021.06.22.449326

Year: 2021

Establishment of bone marrow-derived M-CSF receptor-dependent self-renewing macrophages.

DOI: 10.1038/s41420-020-00300-3

Year: 2020

MEIOSIN Directs the Switch from Mitosis to Meiosis in Mammalian Germ Cells.

DOI: 10.1016/j.devcel.2020.01.010

Year: 2020

Molecular detection of maturation stages in the developing kidney.

DOI: 10.1016/j.ydbio.2020.11.002

Year: 2020

Mesenchymal-epithelial transition regulates initiation of pluripotency exit before gastrulation

DOI: 10.1242/dev.184960

Year: 2020

The polyol pathway is a crucial glucose sensor in Drosophila

DOI: 10.1101/2020.03.16.993170

Year: 2020

MEAF6 is essential for cell proliferation and plays a role in the assembly of KAT7 complexes.

DOI: 10.1016/j.yexcr.2020.112279

Year: 2020

What Are the Consequences of Genetic Mutations?

What are the Implications of BRCA1 and BRCA2 Mutations for Family Members?

Can Mutations be Repaired?

What are the Treatment Options for Cystic Fibrosis?

Are Synonymous Mutations Considered in Genetic Research?

What is the Delta F508 Mutation?

What Are the Side Effects of PARP Inhibitors?

Why is Populace Genomics Important?

What is Third Generation Sequencing?

What Are the Causes of Transversions?

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